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Managing Myofascial Pain Syndrome

Sorting Through the Diagnosis and Honing Treatment

James M. Daniels, MD, MPH; Tim Ishmael, MD; Robert M. Wesley, MA

THE PHYSICIAN AND SPORTSMEDICINE - VOL 31 - NO. 10 - OCTOBER 2003  Read About Mycoplasma  Part -2

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TABLE 2. Recommended Criteria for Identifying Latent and Active Trigger Points

Essential Criteria

Taut band palpable (if muscle is accessible)

Exquisite spot tenderness of a nodule in a taut band

Patient's recognition of current pain complaint by pressure on the tender nodule (identifies active trigger point)

Painful limit to full passive stretch range of motion


Confirmatory Observations

Visual or tactile identification of local twitch response

Observation of a local twitch response induced by needle penetration of a tender nodule

Pain or altered sensation (in the distribution expected from a trigger point in that muscle) on compression of a tender nodule

Electromyographic demonstration of spontaneous electrical activity characteristic of active loci in the tender nodule of a taut band

HRT = hormone replacement therapy; MI = myocardial infarction; BP = blood pressure; HDL = high-density lipoprotein cholesterol

Adapted from Simmons D, Travell J, Simmons S: Trigger Point Manual, vol 1, ed 2. Williams & Wilkins, 1999, p 132.

 

Trigger points are subdivided into two groups. Active trigger points cause referred pain and usually have predictable patterns specific to each muscle. These trigger points are rarely located where the patient reports the pain. The pain pattern does not usually follow a specific dermatomal pattern. The suspected muscle should be palpated until a band or feeling of tightness is located. This finding is often described as a ropiness or nodularity in the muscle. Clinicians should then apply pressure until they find the "spot of maximum tenderness" along the length of the taut band of muscle fiber.14

When the trigger point is palpated, the patient may localize the discomfort and involuntarily withdraw from contact (jump sign).1 Such a finding is a strong indication of a trigger point. Sustained digital pressure on a trigger point usually evokes the same referred pain pattern that brought the patient into the clinic. Occasionally, anatomically controlled phenomena, such as a change in skin temperature, color, or perspiration, may occur.

The second type of myofascial trigger point that Travell15 describes is the latent trigger point. On exam, the patient may have a nodular area that, while associated with a taut band of muscle, does not reproduce pain. This finding, along with increased muscle tension and a restricted range of motion, separates this particular myofascial trigger point from the tender points that are characteristic of fibromyalgia.

Treating Trigger Points

After identifying the characteristic trigger points of MPS and ruling out other diagnoses, various treatment protocols can be implemented in an office setting.

Trigger point injections. Various compounds have been described16,17 for use in injections, including 3% promethazine-hydrochloride, 0.5% procaine hydrochloride, 1% lidocaine hydrochloride without vasoconstrictors, and 0.25% lidocaine with normal saline. Indications for trigger point injections are a limited number of tender spots coinciding with the patient's complaint that produce the jump sign in response to pressure.18 Several contraindications for injections have been proposed (table 3).18

TABLE 3. Contraindications for Trigger Point Injections

Do Not Inject Patients Who Have:

A bleeding disorder or are on anticoagulant therapy

An eating disorder

Taken aspirin within 3 days

A local or systemic infection

Allergies to local anesthesia

An inordinate fear of needles

The International Association for the Study of Pain has made suggestions for training to identify trigger points and has published recommended standards for injecting trigger points.17 Many experienced primary care physicians are skilled in performing injections, but physicians should be aware of the following caveats:

  • To avoid pneumothorax, never aim the needle at an intercostal space. (Even with proper training and patient informed consent, we do not advocate injection in this area for outpatients.)
  • Use a needle long enough to keep the hub well above the skin surface during injection.
  • Never inject the needle to the hub, because, if the needle breaks, complications can arise.
  • Be aware of the tip of the needle at all times and avoid placing any sideward pressure on the syringe that could bend the needle and deflect the tip.
  • Check for a needle that has a rough tip surface, which may create a "drag" upon injection; the impact of the tip of such a needle produces a fish-hook burr, causing unnecessary bleeding.17 Needles can be replaced if a rough tip is found, even after the syringe is filled.

 

Shiatsu. This technique has many names, including ischemic acupressure and mild therapy. To perform the technique, the clinician uses the thumb to apply a slow, gentle, firm pressure to the trigger point. It is very important that light pressure be applied initially and slowly intensified over approximately 1 minute; application of sudden forceful pressure will actually aggravate the patient's symptoms. A second minute of maximal pressure is applied, at which time the examiner's thumb is slowly released from the trigger point. Clinicians familiar with this technique often describe a feeling of the muscle "giving way" beneath their fingers during this second minute. Once pressure is released, the skin blanches briefly, and a reactive hyperemia follows that may last several hours. This technique has no known complications other than local ecchymosis in some patients.

Spray and stretch. Not all patients can tolerate ischemic acupressure, so some practitioners may employ a spray and stretch technique with vapocoolant. The pain signal sent by the stretched muscle is overridden by a nerve impulse to the posterior horn of the spinal column. Following identification of the trigger point, the physician places the patient in a comfortable, relaxed position. To prepare the patient, physicians may often use a heating pad or moist heat on the area for approximately 5 to 10 minutes. One end of the muscle is anchored either by one of the examiner's hands or by immobilizing the patient's distal appendage. The skin is then sprayed with repeated parallel sweeps of vapocoolant over the length of the muscle in the direction of the pain pattern (figure 2). It is important that the vapocoolant container be held approximately 12 in. (30 cm) from the skin at a 30° angle to the plane of the skin. Parallel sprays are made using unidirectional sweeps over the most tightly stretched muscle fibers and then over the rest of the muscle. Sweeps should be slow and even and cover about 4 in./sec (10 cm/sec). The spray should be overlapped slightly, but no more than two passes should be made over the same area.

 

After the first sweep of spray, pressure is applied to take up muscle slack; this pattern is continued as additional sweeps of spray are applied. The sweeps are extended to cover the referred pain pattern of the muscle. These steps are repeated two or three times until the skin becomes cold to the touch or when range of motion reaches its maximum. Reapplication of heat is followed by several cycles of full range of motion of that muscle. Some physicians suggest using the vapocoolant over a slightly larger area than that of the referred pain pattern. If the patient says the spray is too cold, the dispenser can be held closer to the skin than the usual 12 in. (30 cm). If a colder than usual spray is desired, distance can be increased to 18 in. (46 cm). The skin should be transiently cooled and not the underlying muscle. Passive stretching is gradually applied to get the joint to extend to its full range of motion, but the muscle should not be overstretched. The muscle should then be promptly returned to its shortened length.

Coolant use and ice stroking. In the past, the most widely used vapocoolant was ethyl chloride, but this has been replaced by fluoromethane, which is nontoxic, is nonflammable, and does not irritate the skin. Unfortunately, fluorocarbons have been implicated in degradation of the upper atmosphere ozone layer and will no longer be manufactured. A temporary medical exception was granted for fluoromethane until a suitable substitute is developed, and one such spray is available (Fluori-Methane Spray and Stretch, Gebauer, Cleveland, 1-800-321-9348). The concentrated stream from a vapocoolant dispenser is far superior to the usual diffuse spray from a standard spray can. Physicians should be careful when using fluoromethane. Although the spray will not damage most tissues that are accidentally sprayed, the conjunctiva may be damaged if the spray hits a patient's eye.

Some patients with cold-induced asthma or other respiratory conditions may not tolerate vapocoolant spray near the face unless the clinician covers the patient's nose with a small cloth or hand.This difficulty has led to the development of a technique called ice stroking, which may replace spray for these patients. Water is frozen in a plastic or paper cup with a stirring stick, such as a tongue depressor, placed in the cup to provide a handle to hold the ice. The bottom of the cup is then torn back and an edge of ice is applied to the skin in a unidirectional stroke following the same patterns as for the spray. The patient's skin must remain dry, because dampness alters the rate of change in skin temperature. Some clinicians cover the ice with thin plastic wrap or have an assistant follow along with a small towel to blot the skin. It is imperative to move along at a rate so that the ice cools just the skin and not the underlying tissues.1

These techniques can be easily mastered in a short time. The only contraindications to these methods would be if the patient has Raynaud's phenomenon, cold urticaria, or hypersensitivity to one of the cooling agents.

Battling Underdiagnosis

MPS is a common disorder that may be underdiagnosed. It is imperative that the physician has determined that the patient has no other condition that mimics MPS. Some of the simple techniques described for treating MPS can be easily learned by patients to provide pain relief and restore function without the additional cost of supervised physical therapy and medications.

 

References

  1. Simons DG, Travell JG, Simons LS, et al: Travell & Simons' Myofascial Pain and Dysfunction: The Trigger Point Manual, ed 2. Baltimore, Williams & Wilkins, 1999, p 132
  2. Skootsky SA, Jaeger B, Oye RK: Prevalence of myofascial pain in general internal medicine practice. West J Med 1989;151(2):157-160
  3. Kellgren JH: A preliminary account of referred pains arising from muscle. BMJ 1938;1:325-327
  4. Wolfe F, Smythe HA, Yunus MB, et al: The American College of Rheumatology 1990 criteria for the classification of fibromyalgia: report of the Multicenter Criteria Committee. Arthritis Rheum 1990;33(2):160-172
  5. Han SC, Harrison P: Myofascial pain syndrome and trigger-point management. Reg Anesth 1997;22(1):89-101
  6. Jaeger B, Reeves JL: Quantification of changes in myofascial trigger point sensitivity with the pressure algometer following passive stretch. Pain 1986;27(2):203-210
  7. Wolfe F, Simons DG, Fricton J, et al: The fibromyalgia and myofascial pain syndromes: a preliminary study of tender points and trigger points in persons with fibromyalgia, myofascial pain syndrome and no disease. J Rheumatol 1992;19(6):944-951
  8. Tunks E, McCain GA, Hart LE, et al: The reliability of examination for tenderness in patients with myofascial pain, chronic fibromyalgia and controls. J Rheumatol 1995;22(5):944-952
  9. Mountcastle VB (ed): Medical Physiology, ed 14. St Louis, Mosby-Yearbook, 1980, pp 82-119
  10. Fricton JR, Auvinen MD, Dykstra D, et al: Myofascial pain syndrome: electromyographic changes associated with local twitch response. Arch Phys Med Rehabil 1985;66(5):314-317
  11. Fine PG, Milano R, Hare BD: The effects of myofascial trigger point injections are naloxone reversible. Pain 1988;32(1):15-20
  12. Rachlin ES: History and physical examination for regional myofascial pain syndrome, in Rachlin ES (ed): Myofascial Pain and Fibromyalgia: Trigger Point Management. St Louis, Mosby-Yearbook, 1994, pp 159-172
  13. Simons DG, Travell JG: Myofascial origins of low back pain: 1. Principles of diagnosis and treatment. Postgrad Med 1983;73(2):66-77
  14. Price DD, McGrath PA, Rafii A, et al: The validation of visual analogue scales as ratio scale measures for chronic and experimental pain. Pain 1983;17(1):45-56
  15. Travell J: Identification of myofascial trigger point syndromes: a case of atypical facial neuralgia. Arch Phys Med Rehabil 1981;62(3):100-106
  16. Wolf SL, Minkwitz JA: Topical anesthetics: effects on the Achilles tendon and H-reflexes. 2. Stroke patients. Arch Phys Med Rehabil 1989;70(9):673-677
  17. Fields HL (ed): Core curriculum for professional education in pain: a report of the Task Force on Professional Education of the International Association for the Study of Pain. Seattle, IASP Press, 1995
  18. McClaflin RR: Myofascial pain syndrome: primary care strategies for early intervention. Postgrad Med 1994;96(2):56-73
  19. Chan P: Finger Acupressure: Treatment for Many Common Ailments from Insomnia to Impotence by Using Finger Massage on Acupuncture Points. New York City, Ballantine Books, 1975
  20. Nielsen AJ: Spray and stretch for myofascial pain. Phys Ther 1978;58(5):567-569
  21. Halkovich LR, Personius WJ, Clamann HP, et al: Effect of Fluori-Methane spray on passive hip flexion. Phys Ther 1981;61(2):185-189
  22. Mense S, Simons DG, Russell IJ: Muscle Pain: Understanding Its Nature, Diagnosis, and Treatment. Philadelphia, Lippincott Williams & Williams, 2001, p 299

 


Dr Daniels is associate professor of family and community medicine in Southern Illinois University School of Medicine's family practice residency program and program director of the care sports medicine fellowship program in Quincy, Illinois. He has a certificate of added qualifications in primary care sports medicine. Dr Ishmael is a family practice physician in private practice in Litchfield, Illinois. Mr Wesley is director of research and program development in the department of family and community medicine at the Southern Illinois University School of Medicine in Springfield, Illinois. Address correspondence to James M. Daniels, MD, MPH, 612 N 11th St, Suite B, Quincy, IL 62301;


 

 

 

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