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Autoimmune diseases the real terror threat! Inside your body. Your
own defenses forces have turned against you specially in women of
any age.
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Helicobacter Pylori is a communicable bacteria that
drills its way through the stomach mucosa and attaches
itself to the inside of the epithelia or stomach lining. Our
body has a ingenious way of keeping the immune system from
attacking the food we eat. It isolates the stomach’s content
so the immune system can't see it. The body does this by
coating the stomach with a jelly-like substance and
preventing antibodies from entering through the stomach
lining. The food is then digested and enters the small
intestine. If nature didn't separate the food our immune
systems would see it as a foreign intruder and attack it. |
People suffering from H. Pylori have been cured of
ITP if they
took antibiotics to eradicate H. Pylori. Here is a recently
published study.
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(We have a New treatment protocol written in e-book
above)
Platelets. 2005 Mar;16(2):117-9.
High prevalence of sustained remission of idiopathic
thrombocytopenic purpura after Helicobacter pylori
eradication: a long-term follow-up study.
Veneri D,
Department of Medicine, Haematology, University of
Verona, Verona, Italy.
We studied the effect of Helicobacter pylori (H. pylori)
eradication in 43 consecutive H. pylori-infected
patients with idiopathic thrombocytopenic purpura. H.
pylori was eradicated with antibiotics in 41 of them
(95.3%). The difference between the mean platelet count
before and after therapy was statistically significant
(54.3 x 10(9)/l vs. 119.1 x 10(9)/l; P < 0.001). A
sustained remission was observed in 20 patients (48.8%),
after a median follow-up of 31.2 months. None of the
patients still infected by H. pylori after therapy
reached normal platelet values. The long-term
follow-up confirms the efficacy of H. pylori eradication
in H. pylori-infected ITP patients.
PMID: 15823868 [PubMed - in process]
So one needs to look and eradicate HPylori if you
have ITP. "In conclusion, H pylori infection was
involved in most ITP patients older than 40 years in
Japan, and eradication therapy should be the first line
of treatment in H pylori-positive ITP patients."Int
J Hematol. 2005 Feb;81(2):162-8.
What about antibiotics and Neurology
1: Lancet Neurol. 2004 Dec;3(12):744-51.
The promise of minocycline in neurology.
Yong VW,
Department of Neurosciences, University of Calgary, Alberta, Canada.
The capacity of minocycline to alleviate disease for
several neurological disorders in animals is
increasingly being recognised. Indeed, that one drug
alone can attenuate the severity of disease in stroke,
multiple sclerosis, spinal-cord injury, Parkinson's
disease, Huntington's disease, and amyotrophic lateral
sclerosis is astounding. In this review, we describe the
evidence for the efficacy of minocycline in several
animal models of neurological disease, discuss the
mechanisms by which minocycline affects a range of
neurological diseases with diverse causes, and introduce
the emerging investigation of minocycline in clinical
neurology. The encouraging results of minocycline in
experimental neurology bode well for its therapeutic use
in human neurological diseases.
PMID: 15556807 [PubMed - indexed for MEDLINE]
Ann Neurol. 2004 May;55(5):756.
Minocycline reduces gadolinium-enhancing magnetic
resonance imaging lesions in multiple sclerosis.
Metz LM,
Wiad Lek. 2003;56(11-12):591-3.
[Infectious endocarditis in a patient with multiple
sclerosis--case report]
Prystupa A, Mosiewicz J.
Katedry i Kliniki Chorob Wewnetrznych Akademii Medycznej
Multiple sclerosis is chronic demyelination disease
associated with complex immunological disorders,
resulting in increased susceptibility to different
infections. A case of woman, aged 40 with multiple
sclerosis who was admitted to the Internal Medicine Ward
because of severe general state, fever of seven-day
duration and systolic murmur in apex area is discussed
in this paper. Clinical status and performed
diagnostics, among others; echocardiography, blood
cultures, abdominal ultrasonography, allowed to diagnose
infective endocarditis caused by Enterococcus faecalis.
Antibiotics as follows: augmentin, cefuroxim, cefotaxim
and vancomycin were administered parenterally. As a
result of the treatment normalization of temperature and
complete recovery were obtained.
PMID: 15058172 [PubMed - indexed for MEDLINE]
1: Am J Phys Med Rehabil. 1995
Nov-Dec;74(6):415-8.
Role of bacterial infection in exacerbation of multiple
sclerosis.
Rapp NS,
Physical Medicine , William Beaumont Hospital, Royal
Oak, Michigan 48073, USA.
One hundred consecutive patients admitted to the
hospital with a diagnosis of exacerbation of multiple
sclerosis were evaluated for an infectious process. All
patients received a complete blood count, urinalysis,
urine culture with susceptibility studies, blood
cultures, and a chest x-ray at the time of admission. A
control group of 55 patients carrying the diagnosis of
multiple sclerosis but without symptoms of neurologic
decline were also studied. Thirty-five percent of
patients experiencing exacerbation of their disease were
identified as having a significant bacterial infection
compared with 11% in the control group with quiescent
disease. These results were significant with a P value
of < 0.001. When presumptive viral and bacterial
infections diagnosed before admission were included,
almost 50% of patients could have had an exacerbation of
their disease in response to an infectious process.
Bacterial infection might well play a role in
precipitating relapse in multiple sclerosis as well as
influencing treatment.
PMID: 8534384 [PubMed - indexed for MEDLINE]
1: Neurology. 2004 Jul 13;63(1):179-80.
Improvement of a CIDP associated with hepatitis C
virus infection using antiviral therapy.
Corcia P, Barbereau D, Guennoc AM, de Toffol B, Bacq Y.
Department of Neurology, CHRU Bretonneau, Tours, France.
A 57-year-old man with chronic inflammatory
demyelinating polyneuropathy associated with hepatitis C
virus infection was treated successfully with the
combination of peginterferon-alpha-2b and ribavirin.
Viral eradication was confirmed during the 4th week of
treatment and was followed 3 weeks later by neurologic
improvement. The patient resumed normal activity 1 year
after the therapy was completed.
PMID: 15249636 [PubMed - indexed for MEDLINE]
1: J Peripher Nerv Syst. 2004 Jun;9(2):98-103.
Chronic inflammatory demyelinating polyneuropathy
after Campylobacter jejuni infection mimicking
vasculitic mononeuritis multiplex in a diabetic.
Rajabally YA, Sarasamma P, Abbott RJ.
Department of Neurology, University Hospitals of
Leicester, Royal Infirmary, Leicester, UK.
Chronic inflammatory demyelinating polyneuropathy (CIDP)
is a clinically heterogeneous disorder. Recent reports
suggest that CIDP could be more frequent in diabetics.
We report here a case of CIDP presenting as mononeuritis
multiplex with accompanying cranial nerve involvement in
an insulin-dependent diabetic who presented a preceding
Campylobacter jejuni infection and misleading skin
lesions. Treatment with intravenous immunoglobulins was
successful. This case suggests that CIDP should be
considered as a potential diagnosis in all diabetics
with localized, acute or sub-acute onset, and multiple,
or overlapping mononeuropathies.
PMID: 15104697 [PubMed - indexed for MEDLINE]
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